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A neuron sending a signal (i.e., a presynaptic neuron) releases a chemical called a neurotransmitter, which binds to a receptor on the surface of the receiving (i.e., postsynaptic) neuron. Neurotransmitters are released from presynaptic terminals, which may branch to communicate with several postsynaptic neurons.
Nodes of Ranvier are gaps in the myelin along the axons; they contain sodium and potassium ion channels, allowing the action potential to travel quickly down the axon by jumping from one node to the next.
The sodium-potassium pump maintains the resting potential of a neuron. This pump keeps the concentration of sodium outside the cell greater than the concentration inside the cell while keeping the concentration of potassium inside the cell greater than the concentration of potassium outside the cell.
What does the movement of sodium ions across the membrane do to the membrane potential near the open embedded protein? The membrane potential becomes more positive.
What advantage does the organism have when there is a minimum signal required that may have influenced the evolution of this mechanism? organisms receive alot of sensory input.
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When neurons communicate, the neurotransmitters from one neuron are released, cross the synapse, and attach themselves to special molecules in the next neuron called receptors. Receptors receive and process the message, then send it on to the next neuron.
Action potentials travel down a single neuron cell as an electrochemical cascade, allowing a net inward flow of positively charged ions into the axon. Within a cell, action potentials are triggered at the cell body, travel down the axon, and end at the axon terminal.
It takes longer for potassium channels to open. When they do open, potassium rushes out of the cell, reversing the depolarization. Also at about this time, sodium channels start to close. This causes the action potential to go back toward -70 mV (a repolarization).
The membrane depolarizes above a certain threshold potential. Influx of Na+ ions into the neuron can lead to membrane depolarization above the threshold potential; this event triggers the creation of an action potential.
What will happen to the membrane potential in the time follows the closing of the channel? It also depolarize areas further away, but not to the same degree... It returns back to the original. The shapes of some proteins are dependent on the electrical potential in the surrounding area.

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