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Previous studies have revealed that IL-13 is implicated in the pathogenesis of autoimmune diseases, such as systemic lupus erythematosus (SLE), rheumatoid arthritis (RA), systemic sclerosis (SSc), ulcerative colitis (UC), type 1 diabetes (T1D), sjogrens syndrome (SS), etc.
Major Functions of IL-13 This Th2 cytokine has many of the same functions as IL-4 but is absolutely critical for immune defense against nematodes. Autocrine secretion of IL-13 by Hodgkins lymphoma tumor cells may promote expansion of the tumor.
A major function of IL-13 (and IL-4) in the asthmatic airway is to induce chemotaxis of eosinophils to the site of injury. A number of in vitro studies have investigated the role of IL-13-induced chemotaxis and activation of eosinophils.
In the lung, IL-13 is the central mediator of allergic asthma, where it regulates eosinophilic inflammation, mucus secretion, and airway hyperresponsiveness.
Interleukin-13 is made by a type of T lymphocyte. It reduces inflammation by blocking production of cytokines by macrophages. It also increases the number and activity of B lymphocytes. Interleukin-13 made in the laboratory is used as a biological response modifier to boost the immune system in cancer therapy.
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In the lung, IL-13 is the central mediator of allergic asthma, where it regulates eosinophilic inflammation, mucus secretion, and airway hyperresponsiveness.
2021 Form 3596 Paid Preparers Due Diligence Checklist for California Earned Income Tax Credit.
From these observations, it can be concluded that IL-13 is an antiinflammatory cytokine that plays a unique role in the induction and maintenance of IgE production and IgE-mediated allergic responses.
The key pro-inflammatory cytokines are IL-1, IL-6, and TNF-. These cytokines signal via type I cytokine receptors (CCR1) that are structurally divergent from other cytokine receptor types. They are crucial for coordinating cell mediated immune response and play a critical role in modulating the immune system.
Cytokines play an integral role in the coordination and persistence of the inflammatory process in the chronic inflammation of the airways in asthma since they are capable of inducing many of the pro-inflammatory effects characteristic of this disease (table 1).

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