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From these observations, it can be concluded that IL-13 is an antiinflammatory cytokine that plays a unique role in the induction and maintenance of IgE production and IgE-mediated allergic responses.
Previous studies have revealed that IL-13 is implicated in the pathogenesis of autoimmune diseases, such as systemic lupus erythematosus (SLE), rheumatoid arthritis (RA), systemic sclerosis (SSc), ulcerative colitis (UC), type 1 diabetes (T1D), sjogrens syndrome (SS), etc.
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Interleukin (IL)-13 is a T helper 2 (Th2) cytokine that plays important roles in the pathogenesis of asthma. IL-13 induces hypersensitivity of the airways, increased mucous production, elevated serum immunoglobulin (Ig) E levels and increased numbers of eosinophils.
IL-13 is a cytokine secreted by T helper type 2 (Th2) cells, CD4 cells, natural killer T cell, mast cells, basophils, eosinophils and nuocytes. Interleukin-13 is a central regulator in IgE synthesis, goblet cell hyperplasia, mucus hypersecretion, airway hyperresponsiveness, fibrosis and chitinase up-regulation.
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The resolution of inflammation is essential for homeostasis. One mechanism by which this is achieved is the production of various anti-inflammatory molecules in response to an increased proinflammatory environment. Several cytokines including IL4, IL10, and IL13 are well recognized for their anti-inflammatory activity.

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