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Environmental Fate and Behavior Parathion binds tightly to soil particles and has little or no potential for groundwater contamination. Parathion can persist for days or weeks.
In water, methyl parathion is subject to photolysis, with a half-life of 8 days during the summer and 38 days in winter [12]. Breakdown in vegetation: Uptake and metabolism of methyl parathion in plants is fairly rapid.
The rate of MP degradation accelerated docHubly as pH increased. The conversion docHubed 93.5% after 90 min of hydrolysis at a pH of 9, as compared to 69.9% and 49.8% at pH values of 7 and 4, respectively.
Cholinesterases (ChE) are specialized carboxylic ester hydrolases that catalyze the hydrolysis of choline esters. Two types of ChE activity have been identified in mammalian blood and tissues. These are distinguished ing to their substrate specificity and sensitivity to the selective inhibitors.
* Exposure to Methyl Parathion can cause rapid, fatal, organophosphate poisoning with headache, dizziness, blurred vision, tightness in the chest, sweating, nausea and vomiting, diarrhea, muscle twitching, convulsions, coma and death. * There is no evidence that Methyl Parathion causes cancer in animals.
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Antidotes and Other Treatments In the United States, primary antidotal treatment for parathion poisoning involves administration of atropine. In addition, pralidoxime (2-PAM) is indicated for seriously poisoned patients.

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