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Adenosine has no antiarrhythmic effect in catecholamine-dependent reentry or other types of VT. ingly, adenosine termination of VT is diagnostic of cAMP-mediated triggered activity, which is what accounts for most forms of right and left ventricular outflow tract tachycardia.
[15] In the presence of a substrate, ventricular tachycardia is triggered by multiple factors; the most common triggers include myocardial ischemia, hypokalemia, hypomagnesemia, hypocalcemia, sepsis, and metabolic acidosis. Inherited cardiac channelopathies are more common in young individuals.
Although treatment of VT initially required open heart surgery, it has since been replaced by percutaneous ablation, a safe and effective catheter-based therapy to ablate myocardium from either the endocardial or the epicardial surface.
Ventricular tachycardia treatment may include medicines, procedures and devices to control or reset the heart rhythm, and heart surgery. If another medical condition is causing tachycardia, treating the underlying problem may reduce or prevent episodes of a fast heartbeat.
Ventricular tachycardia is a type of irregular heartbeat, called an arrhythmia. It starts in the lower chambers of the heart, called the ventricles. This condition also may be called V-tach or VT.
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0:04 1:48 Tone finally return the patient to the semi-recumbent. Position to complete the maneuver.MoreTone finally return the patient to the semi-recumbent. Position to complete the maneuver.
Ventricular tachycardia is terminated by an effective stimulus (ES) deliv- ered at the same interval of 165 msec after the preceding RV electrogram. RV electrogram. The effectiveness of the premature stimulus in terminating VT was also current-dependent (fig.
A combination of rapid pacing and extrastimuli can terminate VT unresponsive to ventricular extrastimuli or rapid ventricular pacing alone and may avoid acceleration of VT using rapid ventricular pacing at very short cycle lengths for termination.

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