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Received for publication 17 July 1998 and accepted in revised form - ncbi nlm nih

Received for publication 17 July 1998 and accepted in revised form - ncbi nlm nih-2025

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The document discusses the pivotal role of Thymus and Activation-Regulated Chemokine (TARC) in bacteria-induced fulminant hepatic failure in mice. It highlights how TARC, a CC chemokine, recruits IL-4 producing CCR4-expressing CD4+ T cells into the liver during an inflammatory response triggered by Propionibacterium acnes and lipopolysaccharide (LPS). The study demonstrates that neutralizing TARC with a monoclonal antibody can significantly protect against acute liver damage by reducing lymphocyte infiltration and cytokine production associated with liver injury. This suggests that targeting TARC may offer therapeutic potential for managing fulminant hepatic failure.

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Received for publication 17 July 1998 and accepted in revised form - ncbi nlm nih-2025

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