Mechanics of Atherosclerosis 2025

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The document titled 'The Mechanics of Atherosclerosis' by John Huynh and Stacey Coston provides an educational framework for high school students (grades 9-12) to understand the mechanical mechanisms behind atherosclerosis, a leading cause of cardiovascular disease. It outlines objectives, vocabulary, materials, and safety precautions for two laboratory activities: a polymer lab focused on gel fabrication and a blood flow lab simulating circulatory system dynamics. The content emphasizes the relationship between arterial stiffness, disturbed blood flow, and the progression of atherosclerosis, aiming to enhance students' scientific inquiry and analytical skills.
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As plaque builds up, your artery wall grows thicker and harder. This hardening of the arteries is usually a silent process in the early stages. You may not notice symptoms for a long time. But eventually, as the plaque grows, the opening (lumen) of your artery narrows, leaving less room for blood to flow.
Atherosclerosis is a lipoprotein-driven disease that leads to plaque formation at specific sites of the arterial tree through intimal inflammation, necrosis, fibrosis, and calcification.
The basic mechanisms of atherosclerosis involve lipid accumulation and immune activation in the vascular wall (see Fig. 4.1).
Atherosclerosis is initiated by endothelium activation and, followed by a cascade of events (accumulation of lipids, fibrous elements, and calcification), triggers the vessel narrowing and activation of inflammatory pathways.
Atherosclerosis is associated with inflammatory processes in the endothelial cells of the vessel wall associated with retained low-density lipoprotein (LDL) particles. This retention may be a cause, an effect, or both, of the underlying inflammatory process.

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Atherosclerosis is a chronic inflammatory disease in which there is a build up of plaques inside arteries. These plaques are principally composed of lipids that induce an inflammatory reaction causing turbulent flow with atheroscl cardiovascular disease (ASCVD) as a result.

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by S Jebari-Benslaiman 2022 Cited by 764 Atherosclerosis is initiated by endothelium activation and, followed by a cascade of events (accumulation of lipids, fibrous elements, and calcification),

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