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Impairment of L-type Ca2+ Channel-Dependent Forms of
The extracellular matrix glycoprotein tenascin-C (TN-C) has been suggested to play important functional roles during neural development, axonal regeneration
NAD+ and NADH in brain functions, brain diseases and
by W Ying 2007 Cited by 140 It has been reported that NADH can also affect ryanodine-sensitive Ca2+ channels: Hypoxia-induced increases in NADH can directly increase Ca2+ release from IP3
by MR Evers 2002 Cited by 187 Impairment of L-type Ca2+ channel-dependent forms of hippocampal synaptic plasticity in mice deficient in the extracellular matrix glycoprotein tenascin-C.
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