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The nephrons in the kidneys are supplied with a dense network of blood vessels, and high volumes of blood flow through them. Over time, uncontrolled high blood pressure can cause arteries around the kidneys to narrow, weaken or harden. These damaged arteries are not able to deliver enough blood to the kidney tissue.
But there are subsets of patients whose hypertension is difficult to control or who have additional cardiovascular disease. There are also those with changes to their heart or kidneys as a result of high blood pressure. Those patients should see a cardiologist or, if appropriate, a nephrologist, Dr. Giaimo says.
In the phrase renal hypertension, renal relates to your kidneys and hypertension means high blood pressure. This condition happens when the arteries that transport blood to your kidneys get smaller. Complications of renal hypertension include heart attack, aneurysm and stroke.
Some of the most common conditions that a nephrologist may treat include: Diabetic and other kidney diseases. High blood pressure. Kidney failure.
A cardiologist is a healthcare provider who can treat chest pain, high blood pressure and heart failure, as well as problems with your heart valves, blood vessels and other heart and vascular issues.
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Over time, the effects of high blood pressure may cause other symptoms related to your heart and vascular health. It is recommended that a specialist, a cardiologist monitors your care. A cardiologist has special training in identifying, preventing, and treating diseases of the heart and blood vessels.
Along with blood pressure, pulse pressure can also provide insight into your heart health. In some cases a low pulse pressure (less than 40) may indicate poor heart function, while a higher pulse pressure (greater than 60) may reflect leaky heart valves, often as a result of age-related losses in aortic elasticity.
Hypertensive nephropathy (HTN) or hypertensive nephrosclerosis is a kidney disease associated with chronic high blood pressure. HTN is the second most common cause of ESRD [137]. Although podocyte injury is relevant to HTN pathogenesis, human evidence is lacking.

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