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The gate control theory of pain states that the transmission of pain signals can be modulated at the spinal cord level, by non-painful inputs as well as descending signals from the brain. Non-painful inputs typically close the gate to painful inputs, reducing pain. Pain sensation is mediated by pain receptors, or nociceptors, which are essentially nerve endings of first-order neurons in the pain pathway. The axons of these neurons form small-diameter nerve fibers of 2 types: fast fibers A-delta responsible for the initial sharp pain perceived at the time of injury, and slow C-fibers responsible for a dull, longer-lasting pain. First-order neurons travel by way of spinal nerves to the spinal cord, where they synapse with second-order neurons, which, together with third-order neurons, carry pain signals to the brain. However, ing to the gate theory of pain, perception of pain is not simply due to stimulation of nociceptors. There is a so-called nerve
